Search results for "congenital neutropenia"

showing 5 items of 5 documents

Altered chemotactic response to CXCL12 in patients carrying GATA2 mutations.

2015

Abstract GATA2 deficiency—formerly described as MonoMAC syndrome; dendritic cells, monocytes, B cells, and natural killer cell deficiency; familial myelodysplastic syndrome/acute myeloid leukemia; or Emberger syndrome—encompasses a range of hematologic and nonhematologic anomalies, mainly characterized by monocytopenia, B lymphopenia, natural killer cell cytopenia, neutropenia, immunodeficiency, and a high risk of developing acute myeloid leukemia. Herein, we present 7 patients with GATA2 deficiency recruited into the French Severe Chronic Neutropenia Registry, which enrolls patients with all kinds of congenital neutropenia. We performed extended immunophenotyping of their whole blood lymph…

0301 basic medicineAdultMaleReceptors CXCR4AdolescentLymphocyteT-LymphocytesImmunologyMonocytopeniaBiologyNatural killer cell03 medical and health sciencesYoung AdultImmunophenotypinghemic and lymphatic diseasesmedicineImmunology and AllergyHumansLymphocyte CountCongenital NeutropeniaChildAgedCytopeniaB-LymphocytesGATA2 DeficiencyTraditional medicineChemotaxisCell MembraneMyeloid leukemiaCell Biologymedicine.diseaseCD56 AntigenChemokine CXCL12GATA2 Transcription FactorKiller Cells Natural030104 developmental biologymedicine.anatomical_structureImmunologyMutationFemaleJournal of leukocyte biology
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Congenital neutropenia with retinopathy, a new phenotype without intellectual deficiency or obesity secondary toVPS13Bmutations

2013

Over one hundred VPS13B mutations are reported in Cohen syndrome (CS). Most cases exhibit a homogeneous phenotype that includes intellectual deficiency (ID), microcephaly, facial dysmorphism, slender extremities, truncal obesity, progressive chorioretinal dystrophy, and neutropenia. We report on a patient carrying two VPS13B splicing mutations with an atypical phenotype that included microcephaly, retinopathy, and congenital neutropenia, but neither obesity nor ID. RNA analysis of the IVS34+2T_+3AinsT mutation did not reveal any abnormal splice fragments but mRNA quantification showed a significant decrease in VPS13B expression. RNA sequencing analysis up- and downstream from the IVS57+2T>C…

AdultPathologymedicine.medical_specialtyMicrocephalyNeutropeniaDNA Mutational AnalysisVesicular Transport ProteinsNeutropeniamedicine.disease_causeRetinal DiseasesIntellectual DisabilityGene OrderGeneticsmedicineCongenital Bone Marrow Failure SyndromesHumansObesityCongenital NeutropeniaGenetics (clinical)GeneticsMutationCohen syndromebusiness.industryFaciesSyndromemedicine.diseasePhenotypePedigreeVPS13BPhenotypeMutationFemalebusinessRetinopathyAmerican Journal of Medical Genetics Part A
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Application of whole-exome sequencing to unravel the molecular basis of undiagnosed syndromic congenital neutropenia with intellectual disability

2016

International audience; Neutropenia can be qualified as congenital when of neonatal onset or when associated with extra-hematopoietic manifestations. Overall, 30% of patients with congenital neutropenia (CN) remain without a molecular diagnosis after a multidisciplinary consultation and tedious diagnostic strategy. In the rare situations when neutropenia is identified and associated with intellectual disability (ID), there are few diagnostic hypotheses to test. This retrospective multicenter study reports on a clinically heterogeneous cohort of 10 unrelated patients with CN associated with ID and no molecular diagnosis prior to whole-exome sequencing (WES). WES provided a diagnostic yield o…

Male0301 basic medicinePediatricsmedicine.medical_specialtyNeutropeniaAdolescentNeonatal onsetNeutropenia03 medical and health sciences0302 clinical medicinecongenital neutropenia[ SDV.MHEP ] Life Sciences [q-bio]/Human health and pathologyIntellectual DisabilityIntellectual disabilityGeneticsmedicineCongenital Bone Marrow Failure SyndromesHumansExomeChildCongenital NeutropeniaGenetic Association StudiesGenetics (clinical)Exome sequencingRetrospective Studiesbusiness.industryHigh-Throughput Nucleotide SequencingInfantSyndromemedicine.disease3. Good healthPhenotype030104 developmental biologyCHD2Child Preschool030220 oncology & carcinogenesisCohortEtiologyFemalebusiness[SDV.MHEP]Life Sciences [q-bio]/Human health and pathologyBiomarkersAmerican Journal of Medical Genetics Part A
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Idiopathic neutropenia of infancy: Data from the Italian Neutropenia Registry.

2019

Autoimmune neutropenia of infancy (AIN) is characterized by low risk of severe infection, tendency to spontaneously resolve and typically onset at ≤4-5 years of age; it is due to auto-antibodies whose detection is often difficult. In case of negativity of 4 antineutrophils autoantibody tests, after having excluded ethnic, postinfection, drug induced, or congenital neutropenia, according to the Italian guidelines the patients will be defined as affected by "idiopathic neutropenia" (IN). We describe the characteristics of 85 IN patients enrolled in the Italian neutropenia registry: they were compared with 336 children affected by AIN. The 2 groups were clinically very similar and the main dif…

Malemedicine.medical_specialtyNeutropeniaAutoimmunityNeutropeniaDiagnosis Differential03 medical and health sciences0302 clinical medicineSex FactorsMonocytosisRisk FactorsInternal medicineDiagnosismedicineCongenital Bone Marrow Failure SyndromesHumansRegistriesCongenital NeutropeniaHematologyLeukopeniabusiness.industryAge Factors; Autoimmunity; Congenital Bone Marrow Failure Syndromes; Diagnosis Differential; Female; Humans; Infant; Italy; Leukopenia; Male; Neutropenia; Registries; Risk Factors; Sex FactorsAutoantibodyAge FactorsInfantHematologyLeukopeniamedicine.diseaseSettore MED/38Italy030220 oncology & carcinogenesisAutoimmune neutropeniaDifferentialFemalemedicine.symptomDifferential diagnosisbusiness030215 immunologyAmerican journal of hematology
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Periodontal disease associated to systemic genetic disorders

2007

A number of systemic disorders increase patient susceptibility to periodontal disease, which moreover evolves more rapidly and more aggressively. The underlying factors are mainly related to alterations in immune, endocrine and connective tissue status. These alterations are associated with different pathologies and syndromes that generate periodontal disease either as a primary manifestation or by aggravating a pre-existing condition attributable to local factors. This is where the role of bacterial plaque is subject to debate. In the presence of qualitative or quantitative cellular immune alterations, periodontal disease may manifest early on a severe localized or generalized basis - in s…

agranulocitosis infantil genéticasystemic alterationsEnfermedad periodontalDown syndromePapillon-Lefèvre syndromealteraciones sistémicas:CIENCIAS MÉDICAS [UNESCO]síndrome de Chediak-Higashiperiodontitis due to genetic alterationsperiodontitis por alteraciones genéticashyperimmunoglobulinemia Esíndrome de Marfansíndrome de hiperglobulinemia EMarfan syndromesevere congenital neutropenianeutropenia severa congénitaEhler-Danlos syndromesíndrome de Ehler-DanlosUNESCO::CIENCIAS MÉDICASsíndrome de Papillon-Lefévresíndrome de DownPeriodontal diseaseChediak-Higiashi syndromeinfantile genetic agranulocytosis
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